Type I IFN Exacerbates Disease in Tuberculosis Susceptible Mice by Inducing Neutrophil-mediated Lung Inflammation and NETosis

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Tuberculosis (TB) is a leading cause of mortality due to infectious disease, but the factors determining disease progression are unclear. Bio X Cell antibodies are used to study type I IFN exacerbates disease in tuberculosis susceptible mice by inducing neutrophil-mediated lung inflammation and NETosis.
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Overcoming primary and acquired resistance to anti-PD-L1 therapy by induction and activation of tumor-residing cDC1s

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Neutrophil-induced ferroptosis promotes tumor necrosis in glioblastoma progression

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Bio X Cell Reference Spotlight
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Immunocytokines are a promising immunotherapeutic approach against glioblastoma

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Bio X Cell Reference Spotlight
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Endogenous DEL-1 restrains melanoma lung metastasis by limiting myeloid cell–associated lung inflammation

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Bio X Cell Reference Spotlight
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Crizotinib-induced immunogenic cell death in non-small cell lung cancer

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Bio X Cell Reference Spotlight
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STAT5 promotes accessibility and is required for BATF-mediated plasticity at the Il9 locus

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Bio X Cell Reference Spotlight
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Immunoprophylactic and immunotherapeutic control of hormone receptor-positive breast cancer

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Bio X Cell Reference Spotlight
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IL-20 antagonist suppresses PD-L1 expression and prolongs survival in pancreatic cancer models

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Bio X Cell Reference Spotlight
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Decidual NK Cells Transfer Granulysin to Selectively Kill Bacteria in Trophoblasts

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Bio X Cell Reference Spotlight
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